Northwestern Medicine scientists have shown that the bacteria that cause gonorrhea may have evolved mechanisms to stimulate white blood cells into killing other bacteria, promoting the survival of gonorrhea bacteria in the human reproductive tract.
Northwestern Medicine scientists have revealed age-dependent differences in cellular mechanisms for protection from herpes simplex virus encephalitis.
Jonathan Leis, PhD, professor in Microbiology-Immunology, received a grant to support his research on small molecule inhibitors that block the replication of enveloped viruses such as herpes and HIV.
Northwestern Medicine scientists demonstrated the ability of a protein, Cas2, in Legionella pneumophila to cleave nucleic acids resulting in increased infectivity in amoebae, its host organism and transmission vehicle for human infection of Legionnaires’ disease.
Northwestern Medicine scientists identified bacteria genes and key factors that are required for host colonization in squid, which may lead to better understanding how humans develop symbiotic relationships with beneficial bacteria.
Northwestern Medicine scientists created a more objective, precise and quicker way to test the effectiveness of multiple sclerosis drugs that may promote the repair of myelin, a protective sheath on neurons.
Northwestern Medicine scientists uncovered a molecular mechanism behind the regulation of the immune system in the gut.
Scientists used a new technique, fast photochemical oxidation of proteins, to better understand how enveloped viruses infect host cells.
Hyewon Phee, PhD, assistant professor in Microbiology-Immunology, showed that a lack of the protein Pak2 in immune cells may lead to immunodeficiency in patients.
Celeste Mallama, a fourth-year graduate student, studies how the bacterium that causes Legionnaires’ Disease infects host cells.