Northwestern Medicine has launched the Human Longevity Laboratory, a longitudinal, cross-sectional study that will investigate the relationship between chronological age and biological age and validate interventions that may reverse or slow down the processes of aging.
A new Northwestern Medicine study has identified short strands of toxic RNAs that contribute to brain cell death and DNA damage in Alzheimer’s and aged brains.
Decreased activity of a specific signaling pathway in the brain vessels of aging mice and humans was linked to a decline in vascular function and subsequent neurodegeneration, according to a recent Northwestern Medicine study published in the Journal of Clinical Investigation.
Since the COVID-19 pandemic thrust many providers into telehealth appointments, scientists have been asking questions about the benefits and concerns of telehealth. While telehealth was in use and gathering interest prior to the pandemic, the need for it accelerated during a time when people were encouraged to stay home.
Living near to green spaces were associated with slower biological aging, according to a Northwestern Medicine study published in Science Advances.
Advanced biological aging may increase the risk of depression or anxiety in midlife for older adults, according to a recent study published in Nature Communications.
Attendees gathered on May 11 for Alzheimer Day, an annual event hosted by the Mesulam Center for Cognitive Neurology and Alzheimer’s Disease to showcase dementia and aging research conducted throughout Northwestern and bring those discoveries to the community.
Consuming certain types of alcohol over long periods of time as well as binge drinking both speed up biological aging, according to a recent Northwestern Medicine study.
In a new study, Northwestern investigators used artificial intelligence to analyze data from a wide variety of tissues, and discovered that the length of genes can explain most molecular-level changes that occur during aging.
An inflammatory signaling protein called interleukin-6 has been linked to the progression of acute myeloid leukemia, representing a potential target for future treatments of the disease.