Tag: Microbiology
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Human Stem Cell Model Reveals Mechanisms of Herpes Infection
A team of scientists developed a novel stem cell model to demonstrate that a pathway that protects against herpes simplex virus 1 infection is unique to neurons in the brain.
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Exploring the Mechanisms of Poxvirus Replication
A study published in the journal Cell uncovers how poxviruses take control of a protein complex in order to enhance their replication and counteract an immune response in hosts.
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Finding Links Between Inflammation and Infection
Samantha Genardi, a fourth-year student in the Driskill Graduate Program (DGP), studies cell response to bacterial infection in the laboratory of Chyung-Ru Wang, PhD, professor of Microbiology-Immunology.
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Genetic Mutation Linked to Viral Brain Infections
Mutations in a gene called DBR1 may increase patients’ susceptibility to brain infection, according to a study published in Cell.
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Findings Provide New Insight into Widespread Virus Behind Birth Defects
A new study advances the understanding of how human cytomegalovirus replicates within cells and also reveals potential treatment targets for the common virus.
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Untangling How Epstein-Barr Virus Infects Cells
Northwestern Medicine scientists have discovered a protein that acts as an entry point for the Epstein-Barr virus, providing a potential target for future therapies.
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Molecular Mechanism Behind HIV-Associated Dementia Revealed
Northwestern Medicine scientists showed how enzyme inhibitors might be used to halt a molecular process that can cause neurodegeneration in patients with HIV.
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Herpes Virus Mutant Points Towards New Vaccine Strategy
A newly-designed mutant herpes virus provides a strategy for the development of novel herpes virus vaccines.
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New Insights Into Herpes Virus Could Inform Vaccine Development
Research published in Proceedings of the National Academy of Sciences provides new insights into antibodies that block the Epstein-Barr virus from infecting human cells.
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HIV Infection Hijacks Intracellular Highways
A Northwestern Medicine study found the human immunodeficiency virus uses proteins called diaphanous-related formins to hijack the cytoskeleton of healthy cells.
