Scientists have discovered a unique proteomic signature expressed in the blood of persons living with HIV (human immunodeficiency virus) that is associated with age and higher risk for heart failure, findings that can help identify new therapeutic targets for heart failure among both people living with and without HIV.
The NIH recently funded the five-year SILOS Project, led by principal investigator Michelle Birkett, PhD, which will conduct innovative observational research across five cities across the United States to better understand the social contexts that drive HIV and substance use.
Recent work from the laboratory of Elena Martinelli, PhD, MPH, has discovered how inhibiting an immune cell singling pathway in a model of HIV-1 infection may promote immune responses and decrease viral persistence in conjunction with antiretroviral therapy.
Northwestern investigators, faculty, students and community partners came together to share and celebrate global health research, education and outreach as part of the Robert J. Havey, MD Institute for Global Health’s 12th annual Global Health Day.
Naturally occurring variations near the human gene CHD1L may be linked to lower HIV-1 viral load in people of African ancestry, according to a new international, multicenter study published in Nature.
Northwestern Medicine scientists have discovered how HIV hijacks intracellular processes to proliferate and contribute to neurodegeneration, according to a new study published in Nature Communications.
The PrEP4Teens initiative received over $300,000 from from the Chicago Department of Public Health, Alphawood Foundation, Northwestern University Clinical and Translational Sciences Institute, and the Third Coast Center for AIDS Research.
Northwestern Medicine scientists have identified a new compound which could inform future HIV cure strategies, according to research published in Science Advances.
Brian Mustanski, PhD, has been named director of the Third Coast Center for AIDS Research.
Therapeutically inhibiting a metabolic regulator in monocytes unexpectedly increased inflammatory signaling, revealing mechanisms that could inform new strategies to treat chronic inflammatory disorders, according to a recent study.