Miller Named Distinguished Fellow of American Association of Immunologists
Stephen Miller, PhD, the Judy Gugenheim Research Professor of Microbiology-Immunology, has been named an American Association of Immunologists’ 2022 Distinguished Fellow.
Browsing: "Immunology"
Respiratory Virus Reprograms Airway Epithelial Cells
Respiratory syncytial virus infection during infancy results in metabolic reprogramming in epithelial cells lining the airway, according to a recent study.
Investigating Mechanisms Behind Early HIV-1 Infection
Northwestern Medicine investigators have discovered that a microtubule regulatory protein inhibits early HIV type 1 (HIV-1) infection.
Hypoxic Inflammation Blunts Repair After Heart Attack
Inefficient cardiac repair after heart attacks is partially driven by a maladapted response to a low oxygen environment by immune cells, according to a study published in the Journal of Experimental Medicine.
Study Explores Possible Therapeutic Target for SARS-CoV-2
Inhibiting a specific protein complex in SARS-CoV-2, the virus that causes COVID-19, may be a promising therapeutic target for treating the disease, according to a Northwestern Medicine study.
Miller Receives Biomaterials Award for Celiac Disease Nanoparticle Therapy
Stephen Miller, PhD, received the 2021 Technology Innovation and Development Award from the Society For Biomaterials for work on a nanoparticle that induces gluten tolerance in celiac disease.
Inflammation Protection May Be Critical to Treating MS
Prolonging a cellular defense response to inflammation could help regenerate the protective coating of axons that is degraded in diseases such as multiple sclerosis, according to a recent study.
Immune Receptor Linked to Chronic Heart Transplant Rejection
The AXL immune cell receptor has been linked to cardiac allograft vasculopathy, a thickening of vessel walls in transplanted hearts years after implantation, according to a recent study.
Changes in Macrophages Drive Vulnerability to Lung Infections
An age-related accumulation of a signaling molecule may be one culprit behind older adults’ vulnerability to viral pneumonias, according to a recent study.
Viral Protein Rewires B-Cells to Promote Infection
A viral protein of the Epstein-Barr virus previously thought to mimic immune cell receptor signaling actually rewires intracellular signaling in infected cells, promoting viral cell survival and proliferation.